BAD article in the File section
YEah - Deception is the right word. The article is full of truth and
total fiction all woven together.
How is the reader supposed to separate them?
I’d remove this from the files if I was the owner of the list.
the author needs to read a scientific explanation of what is happening
wit the pancreas so as to know what Type 2 diabetes is about. Bernstein
has a good description in his book.
It is NOT as this author says:
"Type II diabetes results from ineffective insulin."
That’s nonsense. There is no such thing as ineffective insulin.
Insulin resistance USUALLY but not always is part of diabetes type 2. I
have type 2 without any insulin resistance. Insulin resistance can be
fixed. That still leaves yo with a pancreas unable to STORE (and often
unable to make) enough insulin. This author apparently forgot about that
- or didn’t know about it.
So his article is totally misleading!
Type 2 diabetes is not the same as insulin resistance. You can have IR
without type 2 diabetes and you can have type 2 diabetes without IR.
Diabetes has to do with pancreas function not insulin resistance.
If you are insulin resistant IN ADDITION or as a trigger for diabetes -
that is separate from the pancreas damage which you have to have to be
diagnosed type 2.
Otherwise you just get a IR diagnosis.
The author has other crazy notions. For example he says:
<<I t would appear quite appropriate to define Type II diabetes more
fundamentally as a failure of the body to metabolize fats and oils
properly. This failure results in a loss of effectiveness of insulin and
in the consequent failure to metabolise carbohydrates.>>
Where does he invent that from? It has no relationship to how fats,
insulin, or carbohydrates are processed in the system!
The body metabolizes fats just fine: If you eat junk fat like trans fat
and saturate fat you will get what you deserve and if you eat good fats
like extra virgin olive oil and fish oil, you will also get what you
deserve:-)
Fats do not affect blood sugar, insulin or insulin sensitivity.
The guy is full of it!
The body fat that is dangerous comes from eating too many carbs. It does
not even come from eating saturated fat. We burn that - first - as
energy:-))) The carbs we eat are stored up front as fat - not the fats!
> I highly recommend this article.
I highly recommend it be removed from the files section for the
completely incorrect statements in it and thus the misleading ideas it
contains.
> Gives a powerful
> persective why you do not want to add insulin to your current program,
And which is completely wrong!
Responsible use of insulin by type 2 diabetics is perhaps the single
best way to rest your pancreas, and allow it to recover functional beta
cells (the ones that make insulin). That’s true because the only
approach that works is one that KEEPS the blood sugar down - not one
that raises it "slowly" or that lowers it later.
Read Dr Bernstein’s diabetes Solution" It contains real science and
metabolism detail then needs no judgement - just facts on how the system
works.
Not this supposition and mis-statement of what happens in the body in
this awful article in the files. I’m sure the author meant well but he
sure did not know his cell or body metabolism. Per the file the author
"Thomas Smith is a reluctant medical investigator". Unfortunately he has
no credentials and it shows - it’s like practising medicine without them
- except it’s a free speech world and who gets hurt doesn’t count when
it’s unintentional.
Namaste,
Irene
August 20th, 2006 at 5:19 am
> Type 2 diabetes is not the same as insulin resistance. You can have IR
> without type 2 diabetes and you can have type 2 diabetes without IR.
>
Irene…. I asked about this a very long time ago on this list (before you
were on it), and was told that IR and T2 are the same thing. My doctor uses the
words interchangeaby as if they are the same thing as well. So is there a
way to tell which one you are?
Kady
[Non-text portions of this message have been removed]
August 21st, 2006 at 12:40 pm
> Irene…. I asked about this a very long time ago on this list (before you
> were on it), and was told that IR and T2 are the same thing.
Not true. However before the function of the pancreas was well
understood (as explained well by Bernstein in his book) this was the
thinking in some circles.
Diabetes has to do with pancreas damage. A healthy pancreas will
manufacture insulin in its beta cells and will also STORE insulin to
have it ready to use at a meal.
In a diabetic, there is no insulin storage, and the function of beta
cells is compromised as well to a larger or lesser extent (totally
compromised in type 1, partly compromised in type 2).
MOST but not all type 2 diabetics ALSO have insulin resistance and
the IR may have triggered the pancreas damage.
One can overcome the IR with a super low carb diet but that will not fix
the diabetes if it is present.
> My doctor uses the
> words interchangeaby as if they are the same thing as well.
:-))
He probably learned that in medical school which is probably some years
ago? Maybe he needs to get a copy of Bernstein’s book or read up on
pancreas issues in diabetes :-))
> So is there a
> way to tell which one you are?
Yes.
IR:
Overweight people are *usually* IR, as the fat tissue is not inert, it
produces cortisol and unless you are VERY low carb diet, there will be
too much sugar in the blood due to carbs eaten added to cortisol making
more sugar. So this constant sugar in the blood causes IR.
There are various tests to measure it. For example your fasting insulin
will be high if you are IR. Mine used to be high (48) but is now near
the bottom of normal range (7).
Diabetic people (IR or not) will have high blood sugar after meals as
they can not store insulin to help bring it down, and will have a high
fasting glucose. Glucose tests can show this.
Type 1 diabetics will have that high glucose after meals stay high as
they make no insulin at all. Type 2 diabetics will have that high
glucose reduce itself at least somewhat after an hour or two as they
usually can still make at least some insulin.
A glucometer used after meals can show what is going on with this.
It is common to be type 1 diabetic and not IR. But it is unusual to be
type 2 diabetic and not IR, as in my case. That’s because it requires a
very low carb diet and/or insulin - and I used both - to keep the blood
sugar low enough long enough to get rid of the IR and for the muscles
not to feel like they are drowning in sugar.
But diabetes is still with me. My pancreas still does not store insulin
as a normal non-diabetic does, so my glucose goes up after meals and my
pancreas also still does not make as much insulin as I’d like after a
meal. So I either have to still eat low carb or use insulin or both.
Does that help?
Namaste,
Irene
August 21st, 2006 at 11:06 pm
>was told that IR and T2 are the same thing. My doctor uses the words
interchangeaby<
Sorry, Kady,
But you can have Type 2 *without* IR (fairly rare, but I have friends who
control with diet and exercise alone), and you can have IR for years before you
become diabetic (as was the case with me).
My pancreas was able to produce enough insulin to control my bgs for 20+ years
(it "sputtered" at times, but it managed), before I reached the tip-over point
at age 49 and was dx’ed.
It’s unfortunate, but I have heard much incorrect information from doctors in
the last 13 years. One even told me that I needed to eat *more* sugar.
Dianne