Re: Does whey protein increase insulin sensitivity or production?

As I was watching CNN tonight, I saw part of a message scrolled across the
bottom of the screen. It said something about taking whey with a high
carbohydrate meal increased insuling secretion. I did miss part of the
scrolling
message. Does anyone on this list know anything about this. Helene

2 Responses to “Re: Does whey protein increase insulin sensitivity or production?”

  1. Dave Black Says:

    A high-carbohydrate meal taken without the whey will increase insulin
    secretion. How about whey without the high carbs? Do you think that
    just perhaps one of the healthiest foods one could possibly eat could
    be under attack because it can be used as a cntral part of a program
    to reverse diabetes type II? My instinct tells me yes.

    The first abstract is the one we’re looking for; it says: "Whey
    proteins have insulinotropic effects and reduce the postprandial
    glycemia in healthy subjects. The mechanism is not known, but
    insulinogenic amino acids and the incretin hormones seem to be
    involved."

    The mechanism is in fact known. Anti-aging research using amino acids
    that was published back in 1981 established that certain amino acids
    elevate HGH, insulin, and IGF-1. Insulin and IGF-1 are strong growth

    factors, and both are necessary to survival. The key to understanding
    health or illness is not the burst of healing power you get from
    triggering their release, but the ambient "resting" levels of insulin
    that we’re trying to keep down between the bursts.

  2. Dave Black Says:

    Dietary whey protein results in increased glycogen storage in the
    liver and less circulating glucose. This is obviously good. I think
    this research publishd in 2005 puts the concept that "whey is bad"
    for a diabetic to rest.

    Here’s the quote:
    "…results demonstrate that the whey protein diet in exercise-
    trained rats results in significantly higher levels of liver
    glycogen, because of the combined effects of regulation of rate
    limiting glycolytic and gluconeogenic enzyme activities and
    activation of glycogenesis from alanine via alanine amino-
    transferase."

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